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At blood pH, UPSe nanoprobes were present as self-assembledmicelles with a diameter of 25.3± 1.5 nm and a spherical morphology (left panel, Fig. 2d). HomoFRET-induced fluorescence quenching was responsible for complete silencing of the fluorophores in the micelle state (Fig. 2c)24.

At blood pH, UPSe nanoprobes were present as self-assembledmicelles with a diameter of 25.3± 1.5 nm and a spherical morphology (left panel, Fig. 2d). HomoFRET-induced fluorescence quenching was responsible for complete silencing of the fluorophores in the micelle state (Fig. 2c)24.

We study the impact of disease-related hypoxic stress with aging upon synaptic plasticity, white matter connectivity, and cognitive performance.

Grinnell Lab's Responsible Conduct of Research Discussion Cases

Mitophagy in cardiac disease and aging

Learn about Mechanisms of AOB plasticity

We hy- pothesize that binding of �-lap inside the hydrophobic cavity of cyclodextrins will drive the dynamic equilibrium of �-lap from solid state to the solution state (Fig. 1), thereby increas- ing the drug solubility.

Regulation of calcineurin by RCAN1

Metabolic reprogramming is now firmly established as a hallmark of cancer

We study the cellular, synaptic and molecular mechanisms of synapse and circuit development and how this is regulated by genes implicated in autism and schizophrenia.